GRAVES DISEASE

Introduction: Graves' desease is a type of hyperthyroidism that usually by the widespread activity throughout the thyroid. The term was named after Robert Graves, an Irish physician, was the first of its kind to describe the hyperthyroidism almost a half centuries. This disorder is also threatened by crop, exophthalmos, orange skin and hyperthyroidism.

Types of Graves' disease: the basic amount or provisional forms of disease are: autoimmune diseases, autoimmune diseases of the thyroid desease, thyroid disease and hyperthyroidism.

Causes: Generally is of the opinion to be mediated by an antibody auto-immune reaction, but the main reason for such a response is required to be known. It is considered one of the most common causes of hyperthyroidism. It is by some process in the body immune system normally guards the body against foreign invaders such as bacteria and viruses.
Immune system destroys foreign invaders with antibody producing cells from blood lymphocytes. Color of the immune system can also cause the disease make antibodies than their lymphocytes against their own tissues, promote or harm them. The antibodies in Graves' disease bind to the cell surface and stimulate the thyroid to produce above-thyroid hormones, which ultimately over-acting thyroid.

Diagnosis: a series of signs are guidelines for the diagnosis of disease, such as exophthalmia and non-pitting edema. Goiter by enlarged thyroid gland with other types of hyperthyroidism, but the most common cause is Graves' desease. The major crop is clearly visible, but smaller crop may not be clinically understandable, of course, help X-rays and ultrasound can be traced. The definitive diagnosis requires biopsy.

Symptoms of hyperthyroidism, Graves' disease is usually the same as those from other types of thyroid glands. Eye Disease: Graves' is the only type of thyroid, inflammation of the eyes, swelling of the tissues around the eyes and bulging of the eyes. Skin disease: In rare cases, patients with Graves' desease are to have noticed lumpy reddish thickening of the skin against the shins called pretibial myxedema. Other common symptoms can be very numerous.

Treatment: It includes anti-thyroid drugs such as methimazole or propylthiouracil to reduce thyroid hormone production, radioactive iodine and surgical thyroidectomy ie the removal of the gland, which still claims preoperative treatment of typical drugs.